[1]Walters J R. Have we forgotten about inherited disease//AGBU Pig Genetics Workshop, 2010: 27-28. [2]孙淑东, 殷凤振, 刘文明, 来长海. 仔猪腹股沟阴囊疝的预防及治疗措施. 山东畜牧兽医, 2011, 32(8): 30-31. Sun S D, Yin F Z, Liu W M, Lai C H. Piglets scrotal/inguinal hernia prevention and treatment measures. Shandong Journal of Animal Husbandry and Veterinary Science, 2011, 32 (8): 30-31. (in Chinese)[3]Chang K S, Chang J H. Animal models of pediatric surgical diseases. Pediatric Surgery International 1994, 9(5/6): 307-322. [4]Gatphayak K, Chongkasikit N, Charoensook R, Laenoi W, Vearasilp T, Sardsud V, Knorr C, ter Meulen U, Brenig B. Present situation of porcine hernia inguinalis/scrotalis in Thailand. The Global Food & Product Chain-Dynamics, Innovalions, Conficts, Strategies, Tropentag Dctober 11-13, 2005. [5]Grindflek E, Moe M, Taubert H, Simianer H, Lien S, Moen T. Genome-wide linkage analysis of inguinal hernia in pigs using affected sib pairs. BMC Genetics, 2006, 7(1): 25. [6]Ding N S, Mao H R, Guo Y M, Ren J, Xiao S J, Wu G Z, Shen H Q, Wu L H, Ruan G F, Brenig B. A genome-wide scan reveals candidate susceptibility loci for pig hernias in an intercross between White Duroc and Erhualian. Journal of Animal Science, 2009, 87(8): 2469-2474. [7]Du F X, Mathialagan N, Dyer C, Grosz M, Messer L, Clutter A, Wang T, Lohuis M, Byatt J. Mapping genes affecting scrotal hernia condition in domestic pigs. Journal of Dairy Science, 2004, 87: 453-453. [8]Du Z Q, Zhao X, Vukasinovic N, Rodriguez F, Clutter AC, Rothschild M F. Association and haplotype analyses of positional candidate genes in five genomic regions linked to scrotal hernia in commercial pig lines. PloS One, 2009, 4(3): e4837. [9]Tanyel F, Müftüoglu S, Dagdeviren A, Kaymaz F, Büyükpamukçu N. Myofibroblasts defined by electron microscopy suggest the dedifferentiation of smooth muscle within the sac walls associated with congenital inguinal hernia. BJU International, 2001, 87(3): 251-255. [10]Tanyel F C, Erdem S, Büyükpamukçu N, Tan E. Smooth muscle within incomplete obliterations of processus vaginalis lacks apoptotic nuclei. Urologia Internationalis, 2002, 69(1): 42-45. [11]Rodrigues Junior A J, Rodrigues C J, Cunha A C, Jin Y. Quantitative analysis of collagen and elastic fibers in the transversalis fascia in direct and indirect inguinal hernia. Revista do Hospital das Clínicas, 2002, 57(6): 265-270. [12]Rodrigues C J, Yoo J H, Rodrigues Junior A J. Elastin (ELN) gene point mutation in patients with inguinal hernia. Genetics and Molecular Biology, 2006, 29(1): 45-46. [13]Andersson L, Georges M. Domestic-animal genomics: deciphering the genetics of complex traits. Nature Reviews Genetics, 2004, 5(3): 202-212. [14]兰旅涛, 郭源梅, 陈从英, 杨斌, 毛辉荣, 任军, 周利华. 在白色杜洛克× 二花脸 F2 资源家系中定位影响猪 210 日龄 8 个体尺性状的 QTL. 中国农业科学, 2010, 43(15): 3214-3220. Lan L T, Guo Y M, Chen C Y, Yang B, Mao H R, Ren J, Zhou L H. QTL Mapping for eight traits related to porcine body dimensions at 210 days in a White Duroc × Erhualian F2 resource population. Scientia Agricultura Sinica, 2010, 43(15): 3214-3220. (in Chinese)[15]Purcell S, Neale B, Todd-Brown K, Thomas L, Ferreira M A, Bender D, Maller J, Sklar P, De Bakker P I, Daly M J. PLINK: a tool set for whole-genome association and population-based linkage analyses. The American Journal of Human Genetics, 2007, 81(3): 559-575. [16]Druet T, Georges M. A hidden Markov model combining linkage and linkage disequilibrium information for haplotype reconstruction and quantitative trait locus fine mapping. Genetics, 2010, 184(3): 789-798. [17]Zhang Z, Guillaume F, Sartelet A, Charlier C, Georges M, Farnir F, Druet T. Ancestral haplotype-based association mapping with generalized linear mixed models accounting for stratification. Bioinformatics, 2012, 28(19): 2467-2473. [18]Yang Q, Cui J, Chazaro I, Cupples L A, Demissie S. Power and type I error rate of false discovery rate approaches in genome-wide association studies. BMC Genetics, 2005, 6(Suppl 1): 134. [19]Barrett J C, Fry B, Maller J, Daly M. Haploview: analysis and visualization of LD and haplotype maps. Bioinformatics 2005, 21(2): 263-265. [20]Horvath S, Xu X, Laird N M. The family based association test method: strategies for studying general genotype–phenotype associations. European Journal of Human Genetics, 2001, 9(4): 301-306. [21]Klein R J, Zeiss C, Chew E Y, Tsai J Y, Sackler R S, Haynes C, Henning A K, SanGiovanni J P, Mane S M, Mayne S T. Complement factor H polymorphism in age-related macular degeneration. Science, 2005, 308(5720): 385-389. [22]McCarthy M I, Hirschhorn J N. Genome-wide association studies: potential next steps on a genetic journey. Human Molecular Genetics, 2008, 17(R2): R156-R165. [23]Brown M D, Sacks D B. IQGAP1 in cellular signaling: bridging the GAP. Trends in Cell Biology, 2006, 16(5): 242-249. [24]Katoh K, Shibata H, Suzuki H, Nara A, Ishidoh K, Kominami E, Yoshimori T, Maki M. The ALG-2-interacting protein Alix associates with CHMP4b, a human homologue of yeast Snf7 that is involved in multivesicular body sorting. Journal of Biological Chemistry, 2003, 278(40): 39104-39113. [25]Vito P, Pellegrini L, Guiet C, D’Adamio L. Cloning of AIP1, a novel protein that associates with the apoptosis-linked gene ALG-2 in a Ca2+-dependent reaction. Journal of Biological Chemistry, 1999, 274(3): 1533-1540. [26]Mahul-Mellier A-L, Hemming F J, Blot B, Fraboulet S, Sadoul R. Alix, making a link between apoptosis-linked gene-2, the endosomal sorting complexes required for transport, and neuronal death in vivo. The Journal of Neuroscience, 2006, 26(2): 542-549. [27]Avila M, Kirchhoff M, Marle N, Hove H D, Chouchane M, Thauvin‐Robinet C, Masurel A, Mosca‐Boidron A L, Callier P, Mugneret F. Delineation of a new chromosome 20q11. 2 duplication syndrome including the ASXL1 gene. American Journal of Medical Genetics Part A, 2013, 161(7): 1594-1598. [28]Li X, Leo BM, Beck G, Balian G, Anderson G D. Collagen and proteoglycan abnormalities in the GDF-5-deficient mice and molecular changes when treating disk cells with recombinant growth factor. Spine, 2004, 29(20): 2229-2234. |