[1]雷财林, 凌忠专, 王久林, 蒋琬如. 北方稻区稻瘟病菌生理小种变化与抗病育种策略. 作物杂志, 2000(3): 14-15.Lei C L, Ling Z Z, Wang J L, Jiang W R. The variation of pathologic races to Magnaporthe grisea and the rice breeding strategy for blast resistance in region of northern China. Agronomial Magazine, 2000(3): 14-15. (in Chinese)[2]李亚, 刘二明, 戴良英, 李成云, 刘林. 湖南稻瘟病菌群体遗传多样性与病菌致病型的关系. 中国水稻科学, 2007, 21(3): 304-308.Li Y, Liu E M, Dai L Y, Li C Y, Liu L. Genetic diversity among populations as related to pathotypes for Magnaporthe grisea in Hunan Province. Chinese Journal of Rice Science, 2007, 21(3): 304-308. (in Chinese)[3]李宏宇, 鲁国东, 王宗华. 稻瘟菌无毒基因研究进展. 中国生物工程杂志, 2003, 23(6): 27-31.Li H Y, Lu G D, Wang Z H. Avirulence genes of in Magnaporthe grisea. China Biotechnology, 2003, 23(6): 27-31. (in Chinese)[4]Valent B. Rice blast as a model system for plant pathology. Phytopathology, 1990, 80(1): 33-36.[5]Flor H H. Current status of the gene-for-gene concept. Annual Review of Phytopathology, 1971, 9: 275-296.[6]Terauchi R, Yoshida K, Saitoh H, Kanzaki H, Okuyama Y, Fujisaki K, Miya A, Abe A, Tamiru M, Tosa Y. Studying genome-wide DNA polymorphisms to understand Magnaporthe-rice interactions. Australasian Plant Pathology, 2011, 40: 328-334.[7]Liu J L, Wang X J, Mitchell T, Hu Y J, Liu X L, Dai L Y, Wang G L. Recent progress and understanding of the molecular mechanisms of the rice-Magnaporthe oryzae interaction. Molecular Plant Pathology, 2010, 11(3): 419-427.[8]Valent B, Khang C H. Recent advances in rice blast effector research. Current Opinion in Plant Biology, 2010, 13: 434-441.[9]张哲, 姜华, 王艳丽, 孙国昌. 稻瘟菌无毒基因研究进展. 遗传, 2011, 33(6): 591-600.Zhang Z, Jiang H, Wang Y L, Sun G C. Progress on avirulence genes of the rice blast fungus Magnaporthe grisea. Hereditas, 2011, 33(6): 591-600. (in Chinese)[10]Jia Y L, McAdams S A, Bryan G T, Hershey H P, Valent B. Direct interaction of resistance gene and avirulence gene products confers rice blast resistance. The EMBO Journal, 2000, 19(15): 4004-4014.[11]Dawkins R, Krebs J R. Arms race between and within species. Proceedings of Royal. Society of London B: Biological Sciences, 1979, 205: 489-511.[12]Woolhouse M E, Webster J P, Domingo E, Charlesworth B, Levin B R. Biological and biomedical implications of the co-evolution of pathogens and their hosts. Nature Genetics, 2002, 32: 569-577.[13]Paterson S, Vogwill T, Buckling A, Benmayor R, Spiers A J, Thomson N R, Quail M, Smith F, Walker D, Libberton B, Fenton A, Hall N, Brockhurst M A. Antagonistic coevolution accelerates molecular evolution. Nature, 2010, 464: 275-278.[14]Stahl E A, Dwyer G, Mauricio R, Kreitman M, Bergelson J. Dynamics of disease resistance polymorphism at the Rpm1 locus of Arabidopsis. Nature, 1999, 400(6745): 667-671.[15]Kanzaki H, Yoshida K, Saitoh H, Fujisaki K, Hirabuchi A, Alaux L, Fournier E, Tharreau D, Terauchi R. Arms race co-evolution of Magnaporthe oryzae AVR-Pik and rice Pik genes driven by their physical interactions. The Plant Journal, 2012, 72: 894-907.[16]Dai Y T, Jia Y L, Correll J, Wang X Y, Wang Y L. Diversification and evolution of the avirulence gene AVR-Pita1 in field isolates of Magnaporthe oryzae. Fungal Genetics and Biology, 2010, 47: 973-980.[17]Chuma I, Isobe C, Hotta Y, Ibaragi K, Futamata N, Kusaba M, Yoshida K, Terauchi R, Fujita Y, Nakayashiki H, Valent B, Tosa Y. Multiple translocation of the AVR-Pita effector gene among chromosomes of the rice blast fungus Magnaporthe oryzae and related species. PLoS Pathogens, 2011, 7(7): e1002147.[18]Kasetsomboon T, Ngam S K, Sriwongchai T, Zhou B, Jantasuriyarat C. Sequence variation of avirulence gene AVR-Pita1 in rice blast fungus, Magnaporthe oryzae. Mycology Progress, 2012, 12: 867-878.[19]Orbach M J, Farrall L, Sweigard J A, Chumley F G, Valent B. A telomeric avirulence gene determines efficacy for the rice blast resistance gene Pi-ta. The Plant Cell, 2000, 12: 2019-2032.[20]Yoshida K, Saitoh H, Fujisawa S, Kanzaki H, Matsumura H, Yoshida K, Tosa Y, Chuma I, Takano Y, Win J, Kamoun S, Terauchia R. Association genetics reveals three novel avirulence genes from the rice blast fungal pathogen Magnaporthe oryzae. The Plant Cell, 2009, 21: 1573-1591.[21]王建飞, 鲍永美, 李培富, 张红生. 基于无毒基因序列的稻瘟病菌指纹类型与致病型的关系初探. 中国水稻科学, 2006, 20(1): 109-112.Wang J F, Bao Y M, Li P F, Zhang H S. Primary study on correlation between pathotypes and DNA fingerprintings based on avirulence gene sequences of rice blast fungus Magnaporthe grisea. Chinese Journal of Rice Science, 2006, 20(1): 109-112. (in Chinese)[22]兰波, 李湘民, 何烈干. 江西省稻瘟病菌的无毒基因分析. 江西农业大学学报, 2010, 32(2): 271-275.Lan B, Li X M, He L G. Analysis on avirulence genes of Magnaporthe oryzae Barr. in rice from Jiangxi province. Acta Agriculturae Universitatis Jiangxiensis, 2010, 32(2): 271-275. (in Chinese)[23]邱福林, 王和和, 陈洁, 庄杰云, Leung H, 程式华, 吴健. 用于水稻突变体大量筛选的DNA微量快速提取法. 中国水稻科学, 2006, 20(3): 329-332.Qiu F L, Wang H H, Chen J, Zhuang J Y, Leung H, Cheng S H, Wu J. A rapid DNA miniprep extraction method for large-scale rice mut ant screening. Chinese Journal of Rice Science, 2006, 20(3): 329-332. (in Chinese)[24]Farman M L, Eto Y, Nakao T, Tosa Y, Nakayashiki H, Mayama S, Leong S A. Analysis of the structure of the AVR1-CO39 avirulence locus in virulent rice-infecting isolates of Magnaporthe grisea. Molecular Plant-Microbe Interactions, 2002, 15(1): 6-16.[25]Tosa Y, Osue J, Eto Y, Oh H S, Nakayashiki H, Mayama S, Leong S A. Evolution of an avirulence gene, AVR1-CO39,concomitant with the evolution and differentiation of Magnaporthe oryzae. Molecular Plant-Microbe Interactions, 2005, 18(11): 1148-1160.[26]李祥晓, 王倩, 罗生香, 何云霞, 朱苓华, 周永力, 黎志康. 黑龙江省稻瘟病菌无毒基因分析及抗病种质资源筛选. 作物学报, 2012, 38(12): 2192-2197.Li X X, Wang Q, Luo S X, He Y X, Zhu L H, Zhou Y L, Li Z K. Analyzing avirulence genes of Magnaporthe oryzae from Heilongjiang province and screening rice germ plasm with resistance to blast fungus. Acta Agronomica Sinica, 2012, 38(12): 2192-2197. (in Chinese)[27]靳春鹏. 水稻抗瘟基因鉴定及稻瘟病菌无毒基因监测[D]. 长春: 吉林大学, 2009.Ji C P. Identification of rice blast resistance gene and monitoring of avirulence gene on Magnaporthe grisea[D]. Changchun: Jilin University, 2009. (in Chinese) |