[1]McDowell J M, Dangl J L. Signal transduction in the plant immune response. Trends in Biochemical Sciences, 2000, 25(2): 79-82.[2]何培青, 柳春燕, 郝林华, 陈靠山, 李光友. 植物挥发性物质与植物抗病防御反应. 植物生理学通讯, 2005, 41(1): 105-110. He P Q, Liu C Y, Hao L H, Chen K S, Li G Y. Volatile organic compounds and plant defence against pathogenic disease. Plant Physiology Journal. 2005, 41(1): 105-110. (in Chinese)[3]Gundlach H, Müller M J, Kutchan T M, Zenk M. Jasmonic acid is a signal transducer in elicitor-induced plant cell cultures. Proceedings of the National Academy of Sciences of the USA, 1992, 89(6): 2389-2393.[4]Diaz J, ten Have A, van Kan J A. The role of ethylene and wound signaling in resistance of tomato to Botrytis cinerea. Plant Physiology, 2002, 129(3): 1341-1351.[5]Thomma B P, Eggermont K, Tierens K F, Broekaert W F. Requirement of functional ethylene-insensitive 2 gene for efficient resistance of Arabidopsis to infection by Botrytis cinerea. Plant Physiology, 1999, 121(4): 1093-1102.[6]Si-Ammour A, Mauch-Mani B, Mauch F. Quantification of induced resistance against phytophthora species expressing GFP as a vital marker: Beta-aminobutyric acid but not BTH protects potato and Arabidopsis from infection. Molecular Plant Pathology, 2003, 4(4): 237-248.[7]Kachroo P, Shanklin J, Shah J, Whittle E J, Klessig D F. A fatty acid desaturase modulates the activation of defense signaling pathways in plants. Proceedings of the National Academy of Sciences of the USA, 2001, 98(16): 9448-9453.[8]de Torres-Zabala M, Truman W, Bennett M H, Lafforgue G, Mansfield J W, Igea P R, Bogre L, Grant M. Pseudomonas syringae pv. tomato hijacks the Arabidopsis abscisic acid signalling pathway to cause disease. The EMBO Journal, 2007, 26: 1434-1443.[9]Laurent Z, Jean-Pierre M, Brigitte M M. β-aminobutyric acid-induced protection of Arabidopsis against the necrotrophic fungus Botrytis cinerea. Plant Physiology, 2011, 1261(2): 517-523.[10]Thomma B P H J, Nelissen I, Eggermont K, Broekaert W F. Deficiency in phytoalexin production causes enhanced susceptibility of Arabidopsis thaliana to the fungus Alternaria brassicicola. The Plant Journal, 1999, 19(2): 163-171.[11]Pape S, Thurow C, Gatz C. The Arabidopsis PR-1 promoter contains multiple integration sites for the coactivator NPR1 and the repressor SNI1. Plant Physiology, 2010, 154(4): 1805-1818.[12]Canet J V, Dobon A, Roig A, Tornero P. Structure-function analysis of npr1 alleles in Arabidopsis reveals a role for its paralogs in the perception of salicylic acid. Plant Cell Environ, 2010, 33(11): 1911-1922.[13]张红志, 蔡新忠. 病程相关基因非表达子1(NPR1):植物抗病信号网络中的关键节点. 生物工程学报, 2005, 21(4): 511-515.Zhang H Z, Cai X Z. Nonexpressor of pathogenesis-related genes 1 (NPR1): A key node of plant disease resistance signalling network. Chinese Journal of Biotechnology, 2005, 21(4): 511-515. (in Chinese)[14]Attaran E, Zeier T E, Griebel T, Zeier J. Methyl salicylate production and jasmonate signaling are not essential for systemic acquired resistance in Arabidopsis. The Plant Cell, 2009, 21(3): 954-971.[15]Manners J M, Penninckx I A, Vermaere K, Kazan K, Brown R L, Morgan A, Maclean D J, Curtis M D, Cammue B P A, Broekaert W F. The promoter of the plant defensin gene PDF1.2 from Arabidopsis is systemically activated by fungal pathogens and responds to methyl jasmonate but not to salicylic acid. Plant Molecular Biology, 1998, 38(6): 1071-1080.[16]Ochoa-Zarzosa A, Loeza-Angeles H, Sagrero-Cisneros E, Villagomez-Gomez E, Lara-Zarate L, Lopez-Meza J E. Antibacterial activity of thionin Thi2.1 from Arabidopsis thaliana expressed by bovine endothelial cells against Staphylococcus aureus isolates from bovine mastitis. Veterinary Microbiology, 2008, 127(3/4): 425-430.[17]Chan Y L, Prasad V, Sanjaya, Chen K H, Liu P C, Chan M T, Cheng C P. Transgenic tomato plants expressing an Arabidopsis thionin (Thi2.1) driven by fruit-inactive promoter battle against phytopathogenic attack. Planta, 2005, 221(3): 386-393.[18]Veronese P, Chen X, Bluhm B, Salmeron J, Dietrich R, Mengiste T. The BOS loci of Arabidopsis are required for resistance to Botrytis cinerea infection. The Plant Journal, 2004, 40(4): 558-574.[19]Denby K J, Kumar P, Kliebenstein D J. Identification of Botrytis cinerea susceptibility loci in Arabidopsis thaliana. The Plant Journal, 2004, 38(3): 473-486.[20]Veronese P, Nakagami H, Bluhm B, AbuQamar S, Chen X, Salmeron J, Dietrich R A, Hirt H, Mengiste T. The membrane-anchored BOTRYTIS-INDUCED KINASE1 plays distinct roles in Arabidopsis resistance to necrotrophic and biotrophic pathogens. The Plant Cell, 2006, 18(1): 257-273.[21]Canonne J, Marino D, Jauneau A, Pouzet C, Briere C, Roby D, Rivas S. The Xanthomonas type III effector XopD targets the Arabidopsis transcription factor MYB30 to suppress plant defense. The Plant Cell, 2011, 23(9): 3498-3511.[22]Li L, Yu X, Thompson A, Guo M, Yoshida S, Asami T, Chory J, Yin Y H. Arabidopsis MYB30 is a direct target of BES1 and cooperates with BES1 to regulate brassinosteroid-induced gene expression. The Plant Journal, 2009, 58(2): 275-286.[23]Xing J H, Weng Q Y, Hao C C, Jia J, Kou H M, Han J M, Dong J G. T1N6_22 gene is required for biotic and abiotic stress responses in Arabidopsis. Russian Journal of Genetics, 2012, 48(12): 1191-1198.[24]Uknes S, Mauch-Mani B, Moyer M, Potter S, Williams S, Dincher S, Chandler D, Slusarenko A, Ward E, Ryals J. Acquired resistance in Arabidopsis. The Plant Cell, 1992, 4(6): 645-656.[25]Ryals J A, Neuenschwander U H, Willits M G, Molina A, Steiner H Y, Hunt M D. Systemic acquired resistance. The Plant Cell, 1996, 8(10): 1809-1819.[26]Hyung G M, Kristin A L, Eugene P P, Kosma D K, Cooper B R, Park H C, AbuQamar S, Boccongelli C, Miyazaki S, Consiglio F, Chilosi G, Bohnert H J, Bressan R A, Mengiste T, Jenks M A. The Arabidopsis RESURRECTION1 gene regulates a novel antagonistic interaction in plant defense to biotrophs and necrotrophs1. Plant Physiology, 2009, 115(1): 290-305. |