[1]Morasso M I, Grinberg A, Robinson G, Sargent T D, Mahon K A. Placental failure in mice lacking the homeobox gene Dlx3. Proceedings of the National Academy of Sciences of the United States of America,1999, 96(1): 162-167.[2]Beanan M J, Sargent T D. Regulation and function of Dlx3 in vertebrate development. Developmental Dynamics, 2000, 218(4): 545-553.[3]Hwang J, Mehrani T, Millar S E, Morasso M I. Dlx3 is a crucial regulator of hair follicle differentiation and cycling. Development, 2008, 135(18): 3149-3159.[4]Han L, Dias Figueiredo M, Berghorn K A, Iwata T N, Clark-Campbell P A, Welsh I C, Wang W, O’brien T P, Lin D M, Roberson M S. Analysis of the gene regulatory program induced by the homeobox transcription factor distal-less 3 in mouse placenta. Endocrinology, 2007, 148(3): 1246-1254.[5]Choi S J, Roodman G D, Feng J Q, Song I S, Amin K, Hart P S, Wright J T, Haruyama N, Hart T C. In vivo impact of a 4 bp deletion mutation in the DLX3 gene on bone development. Developmental Biology, 2009, 325(1): 129-137.[6]Choi S J, Song I S, Ryu O H, Choi S W, Hart P S, Wu W W, Shen R F, Hart T C. A 4 bp deletion mutation in DLX3 enhances osteoblastic differentiation and bone formation in vitro. Bone, 2008, 42(1): 162-171.[7]Mardaryev A N, Ahmed M I, Vlahov N V, Fessing M Y, Gill J H, Sharov A A, Botchkareva N V. Micro-RNA-31 controls hair cycle-associated changes in gene expression programs of the skin and hair follicle. The FASEB Journal, 2010, 24(10): 3869-3881.[8]van Steensel M A,Happle R,Steljlen P M. Molecular genetics of the hair follicle: the state of the art. Proceedings of the Society for Experimental Biology and Medicine, 2000, 223(1): 1-7.[9]Park G T, Morasso M I. Bone morphogenetic protein-2 (BMP-2) transactivates Dlx3 through Smad1 and Smad4: alternative mode for Dlx3 induction in mouse keratinocytes. Nucleic Acids Research, 2002, 30(2): 515-522.[10]Price J A, Bowden D W, Wright J T, Pettenati M J, Hart T C. Identification of a mutation in DLX3 associated with tricho-dento- osseous (TDO) syndrome. Human Molecular Genetics, 1998, 7(3): 563-569.[11]Haldeman R J, Cooper L F, Hart T C, Phillips C, Boyd C, Lester G E, Wright J T. Increased bone density associated with DLX3 mutation in the tricho-dento-osseous syndrome. Bone, 2004, 35(4): 988-997.[12]Drouilhet L, Lecerf F, Bodin L, Fabre S, Mulsant P. Fine mapping of the FecL locus influencing prolificacy in Lacaune sheep. Animal Genetics, 2009, 40(6): 804-812.[13]Holland M P, Bliss S P, Berghorn K A, Roberson M S. A role for CCAAT/enhancer-binding protein beta in the basal regulation of the distal-less 3 gene promoter in placental cells. Endocrinology, 2004, 145(3): 1096-1105.[14]Park G T, Morasso M I. Regulation of the Dlx3 homeobox gene upon differentiation of mouse keratinocytes. The Journal of Biological Chemistry, 1999, 274(37): 26599-26608.[15]Sambrook J, Russell D W. 黄培堂, 王嘉玺, 朱厚础, 张兆山, 陈慧鹏, 范明, 俞炜源, 贺福初译. 分子克隆实验指南. 3版. 北京: 科学出版社, 2002: 463.Sambrook J, Russell D W. Translated by Huang P T, Wang J X, Zhu H C, Zhang Z S, Chen H P, Fan M, Yu W Y, He F C. Molecular Cloning: A Laboratory Manual. 3rd ed. Beijing: Science Press, 2002: 463. (in Chinese)[16]Schmidt-Ullrich R, Aebischer T, Hulsken J, Birchmeier W, Klemm U, Scheidereit C. Requirement of NF-κB/Rel for the development of hair follicles and other epidermal appendices. Development, 2001, 128(19): 3843-3853.[17]Petersson M, Brylka H, Kraus A, John S, Rappl G, Schettina P, Niemann C. TCF/lef1 activity controls establishment of diverse stem and progenitor compartments in mouse epidermis. The EMBO Journal, 2011, 30(15): 3004-3018.[18]Mecklenburg L, Paus R, Halata Z, Bechtold L S, Fleckman P, Sundberg J P. FOXN1 is critical for onycholemmal terminal differentiation in nude (Foxn1) mice. The Journal of Investigative Dermatology, 2004, 123(6): 1001-1011.[19]Mecklenburg L, Nakamura M, Sundberg J P, Paus R. The nude mouse skin phenotype: The role of Foxn1 in hair follicle development and cycling. Experimental and Molecular Pathology, 2001, 71(2): 171-178.[20]Pantalacci S, Chaumont A, Benoit G, Sadier A, Delsuc F, Douzery E J, Laudet V. Conserved features and evolutionary shifts of the EDA signaling pathway involved in vertebrate skin appendage development. Molecular Biology and Evolution, 2008, 25(5): 912-928[21]颜炳学. 鸡类胰岛素生长因子-I基因单碱基突变对该基因转录、表达以及对生长、屠体性状的影响[D]. 北京: 中国农业大学, 2005.Yan B X. The effects of the SNP in promoter region of IGF-I on transcriptional and expressional level of the gene and on growth and carcass traits in chickens[D]. Beijing: China Agricultural University, 2005. (in Chinese)[22]荣恩光, 王志鹏, 张志威, 杨华, 李辉, 王宁. 中国美利奴羊DLX3基因3′UTR的多态性及其与羊毛品质性状的关联分析. 畜牧兽医学报, 2012, 43(3): 358-367.Rong E G, Wang Z P, Zhang Z W, Yang H, Li H, Wang N. Polymorphism in 3′UTR of DLX3 gene and its association with wool quality traits in Chinese Merino Sheep. Acta Veterinaria et Zootechnica Sinica, 2012, 43(3): 358-367. (in Chinese)[23]Galbraith H. Fundamental hair follicle biology and fine fibre production in animals. Animal, 2010, 4(9): 1490-1509.[24]Rogers G E. Biology of the wool follicle: an excursion into a unique tissue interaction system waiting to be re-discovered. Experimental Dermatology, 2006, 15(12): 931-949. |